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10 Signs and Symptoms of Hypothyroidism 1. Feeling Tired. One of the most common symptoms of hypothyroidism is feeling worn out. 2. Gaining Weight. Unexpected weight gain is another common symptom of hypothyroidism ( 7 ). 3. Feeling Cold. Heat is a byproduct of burning calories. 4. Weakness and ...
Signs of hypothyroidism in women 1. Fatigue. Feeling tired after a long day is normal. 2. Dry hair and skin. Women with hypothyroidism have dry hair and skin. 3. Constipation. The digestive system slows down because of the slow metabolic process. 4. Brain fog. This is a situation where there is ...
The signs and symptoms of hypothyroidism vary, depending on the severity of the hormone deficiency. Problems tend to develop slowly, often over a number of years. At first, you may barely notice the symptoms of hypothyroidism, such as fatigue and weight gain.
The Hypothyroidism Symptoms in women are usually sighted as they age. But sometimes the luck might be complete against a few. Then hypothyroidism symptoms in women are also found in children or young girls. This thyroid condition skyrockets your worries and grounds your confidence. It is mainly attributed to the fact that the main low thyroid symptoms in women are weight gain and hair loss. This unexplained weight gain drastically lowers your confidence levels mainly in women if thyroid ...
Since hypothyroidism shares many of the same symptoms, such as fatigue, erratic periods, and weight gain, it can be hard for women to tease the two issues apart initially, says Dr. Leung.
Freezing your ass off all the time is one... Depression is a symptom many women experience, and it can be hard to recognize that the thyroid might be involved, since depression itself can lead to some of the symptoms of hypothyroidism. If you have hyperthyroidism, your mood might be altered another way.
The thyroid gland, or simply the thyroid, is an endocrine gland in the neck, consisting of two lobes connected by an . It is found at the front of the neck, below the Adam's apple. The thyroid gland secretes thyroid hormones, which primarily influence the metabolic rate and protein synthesis. The hormones also have many other effects including those on development. The thyroid hormones triiodothyronine (T3) and thyroxine (T4) are created from iodine and tyrosine. The thyroid also produces the hormone calcitonin, which plays a role in calcium homeostasis. Hormonal output from the thyroid is regulated by thyroid-stimulating hormone (TSH) secreted from the anterior pituitary gland, which itself is regulated by thyrotropin-releasing hormone (TRH) produced by the hypothalamus. The thyroid may be affected by several diseases. Hyperthyroidism occurs when the gland produces excessive amounts of thyroid hormones, the most common cause being Graves' disease, an autoimmune disorder. In contrast, hypothyroidism is a state of insufficient thyroid hormone production. Worldwide, the most common cause is iodine deficiency. Thyroid hormones are important for development, and hypothyroidism secondary to iodine deficiency remains the leading cause of preventable intellectual disability. In iodine-sufficient regions, the most common cause of hypothyroidism is Hashimoto's thyroiditis, also an autoimmune disorder. In addition, the thyroid gland may also develop several types of nodules and cancer.
Hashimoto's thyroiditis, also known as chronic lymphocytic thyroiditis and Hashimoto's disease, is an autoimmune disease in which the thyroid gland is gradually destroyed. Early on there may be no symptoms. Over time the thyroid may enlarge forming a painless goiter. Some people eventually develop hypothyroidism with its accompanying weight gain, feeling tired, constipation, depression, and general pains. After many years the thyroid typically shrinks in size. Potential complications include thyroid lymphoma. Hashimoto's thyroiditis is thought to be due to a combination of genetic and environmental factors. Risk factors include a family history of the condition and having another autoimmune disease. Diagnosis is confirmed with blood tests for TSH, T4, and anti-thyroid autoantibodies. Other conditions that can produce similar symptoms include Graves’ disease and nontoxic nodular goiter. Hashimoto's thyroiditis is typically treated with levothyroxine. If hypothyroidism is not present some may recommend no treatment while others may treat to try to reduce the size of the goiter. Those affected should avoid eating large amounts of iodine; however, sufficient iodine is required especially during pregnancy. Surgery is rarely required to treat the goiter. Hashimoto's thyroiditis affects about 5% of the population at some point in their life. It typically begins between the ages of 30 and 50 and is much more common in women than men. Rates of disease appear to be increasing. It was first described by the Japanese physician Hakaru Hashimoto in 1912. In 1957 it was recognized as an autoimmune disorder.
Hypothyroidism, also called underactive thyroid or low thyroid, is a disorder of the endocrine system in which the thyroid gland does not produce enough thyroid hormone. It can cause a number of symptoms, such as poor ability to tolerate cold, a feeling of tiredness, constipation, depression, and weight gain. Occasionally there may be swelling of the front part of the neck due to goiter. Untreated hypothyroidism during pregnancy can lead to delays in growth and intellectual development in the baby or cretinism. Worldwide, too little iodine in the diet is the most common cause of hypothyroidism. In countries with enough iodine in the diet, the most common cause of hypothyroidism is the autoimmune condition Hashimoto's thyroiditis. Less common causes include: previous treatment with radioactive iodine, injury to the hypothalamus or the anterior pituitary gland, certain medications, a lack of a functioning thyroid at birth, or previous thyroid surgery. The diagnosis of hypothyroidism, when suspected, can be confirmed with blood tests measuring thyroid-stimulating hormone (TSH) and thyroxine levels. Salt iodization has prevented hypothyroidism in many populations.