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Ringworm isn’t a worm. It’s a skin infection that’s caused by mold like fungi that live on the dead tissues of your skin, hair, and nails. You can get it in any of these places -- and on your scalp. When you get it between your toes, it’s what people call athlete’s foot. If it spreads to your groin, it’s known as jock itch.
Causes. Ringworm is a contagious fungal infection caused by mold-like parasites that live on the cells in the outer layer of your skin. It can be spread in the following ways: Human to human. Ringworm often spreads by direct, skin-to-skin contact with an infected person. Animal to human.
Causes. Ringworm of the scalp is caused by one of several varieties of mold-like fungi called dermatophytes. The fungi attack the outer layer of skin on the scalp and the hair shaft. Ringworm isn't caused by a worm. The common name for the disorder refers to the ring-like or circular appearance of the infection on the skin.
They cause a ring or circle-like effect of dry, flaky skin. Sometimes it does not appear in rings at all, but just as flaky, red, itchy skin, with rough patches. After contact, ringworm can take a few days to a week to appear. Ringworm can be treated with antifungal medication.
It helps to be aware of the causes of ringworm so you can avoid them At the heart of ringworm are dermatophytes, which are fungi that cause skin infections (in this case, ringworm). Dermatophytes are keratinophilic, meaning they use the keratin in your skin as a fuel source. 1 , 2 They can grow on humans, animals or in soil, and are highly contagious once they start spreading. 3
Ringworm, or dermatophytosis, is not a worm, nor is it associated with worms of any kind. In fact, there is a good chance you might have had a type of ringworm and not even known it. In reality, ringworm is a skin infection that is caused by a fungal infection on the surface of the skin.
Microsporum gallinae is a fungus of the genus Microsporum that causes dermatophytosis, commonly known as ringworm. Chickens represent the host population of Microsporum gallinae but its opportunistic nature allows it to enter other populations of fowl, mice, squirrels, cats, dogs and monkeys. Human cases of M. gallinae are rare, and usually mild, non-life-threatening superficial infections.
Microsporum gypseum Microsporum is a genus of fungi that causes tinea capitis, tinea corporis, ringworm, and other dermatophytoses (fungal infections of the skin). Microsporum forms both macroconidia (large asexual reproductive structures) and microconidia (smaller asexual reproductive structures) on short conidiophores. Macroconidia are hyaline, multiseptate, variable in form, fusiform, spindle-shaped to obovate, 7–20 by 30–160 um in size, with thin or thick echinulate to verrucose cell walls. Their shape, size and cell wall features are important characteristics for species identification. Microconidia are hyaline, single-celled, pyriform to clavate, smooth-walled, 2.5–3.5 by 4–7 um in size and are not diagnostic for any one species. The separation of this genus from Trichophyton is essentially based on the roughness of the macroconidial cell wall, although in practice this may sometimes be difficult to observe. Seventeen species of Microsporum have been described; however, only the more common species are included in these descriptions.
Trichophyton tonsurans is a fungus in the family Arthrodermataceae that causes ringworm infection of the scalp. It was first recognized by David Gruby in 1844. Isolates are characterized as the "–" or negative mating type of the Arthroderma vanbreuseghemii complex. This species is thought to be conspecific with T. equinum, although the latter represents the "+" mating strain of the same biological species Despite their biological conspecificity, clones of the two mating types appear to have undergone evolutionary divergence with isolates of the T. tonsurans-type consistently associated with Tinea capitis (particularly in children) whereas the T. equinum-type, as its name implies, is associated with horses as a regular host. Phylogenetic relationships were established in isolates from Northern Brazil, through fingerprinting polymorphic RAPD and M13 markers. There seems to be lower genomic variability in the T. tonsurans species due to allopatric divergence. Any phenotypic density is likely due to environmental factors, not genetic characteristics of the fungus.