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What Are the Causes of Elevated Muscle Enzymes? Enzyme Types. There are certain muscle enzymes that are often measured when a liver enzyme elevation... Conditions. Certain types of hepatitis (viral infection that affects the liver), A, B and C,... Other Causes. Trauma, medications, drugs and ...
Muscle cells contain a variety of enzymes with particularly high concentrations of those involved in generating energy to power the muscles. When muscle cells are damaged, these enzymes leak into the bloodstream and may causes elevated levels. A variety of diseases and conditions can cause high muscle enzyme levels.
Higher levels of CPK can also be seen after trauma, injections into a muscle, muscle disease due to an underactive thyroid, and while taking certain medications such as cholesterol-lowering statin ...
The following are possible causes of elevated levels of muscle enzymes: Trauma or injury. When this occurs, the body will produce certain enzymes quicker... Viral, fungal, and bacterial infections. Other ailments such as sarcoidosis, dermatomyositis, polymositis, hepatitis, and muscular ...
High muscle enzyme levels are the result of damaged muscle tissue releasing enzymes into the blood or liver. Muscle damage can be caused by neuromuscular disorders such as muscular dystrophy. Potential liver and muscle damage may be assessed by examining the enzymes in the blood.
Many causes of high muscle enzymes are relatively harmless. For example, common muscle strains can cause your creatine phosphokinase levels to elevate. Your SGOT levels may also elevate because of any type of muscle damage. Elevated lactate dehydrogenase levels may indicate cell damage of any kind.
Exertional rhabdomyolysis (ER) is the breakdown of muscle from extreme physical exertion. It is one of many types of rhabdomyolysis that can occur, and because of this the exact prevalence and incidence are unclear.
Muscle weakness or myasthenia (my- from Greek μυο meaning "muscle" + -asthenia ἀσθένεια meaning "weakness") is a lack of muscle strength. The causes are many and can be divided into conditions that have either true or perceived muscle weakness. True muscle weakness is a primary symptom of a variety of skeletal muscle diseases, including muscular dystrophy and inflammatory myopathy. It occurs in neuromuscular junction disorders, such as myasthenia gravis. Muscle weakness can also be caused by low levels of potassium and other electrolytes within muscle cells. It can be temporary or long-lasting (from seconds or minutes to months or years).
Rhabdomyolysis is a condition in which damaged skeletal muscle breaks down rapidly. Symptoms may include muscle pains, weakness, vomiting, and confusion. There may be tea-colored urine or an irregular heartbeat. Some of the muscle breakdown products, such as the protein myoglobin, are harmful to the kidneys and may lead to kidney failure. The muscle damage is most often the result of a crush injury, strenuous exercise, medications, or drug abuse. Other causes include infections, electrical injury, heat stroke, prolonged immobilization, lack of blood flow to a limb, or snake bites. Some people have inherited muscle conditions that increase the risk of rhabdomyolysis. The diagnosis is supported by a urine test strip which is positive for "blood" but the urine contains no red blood cells when examined with a microscope. Blood tests show a creatine kinase greater than 1,000 U/L, with severe disease being above 5,000 U/L. The mainstay of treatment is large quantities of intravenous fluids. Other treatments may include dialysis or hemofiltration in more severe cases. Once urine output is established sodium bicarbonate and mannitol are commonly used but they are poorly supported by the evidence. Outcomes are generally good if treated early. Complications may include high blood potassium, low blood calcium, disseminated intravascular coagulation, and compartment syndrome. Rhabdomyolysis occurs in about 26,000 people a year in the United States. While the condition has been commented on throughout history, the first modern description was following an earthquake in 1908. Important discoveries as to its mechanism were made during the Blitz of London in 1941. It is a significant problem for those injured in earthquakes and relief efforts for such disasters often include medical teams equipped to treat survivors with rhabdomyolysis.