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  • Group B Strep Support

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    Group B Strep Support is a national charity based in the United Kingdom.

  • Granadaene

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    Granadaene is the trivial name of a non-isoprenoid polyene that constitutes the red pigment characteristic of Streptococcus agalactiae (group B streptococcus).

  • Group B streptococcal infection

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    Group B streptococcus infection, also known as Group B streptococcal disease, is the infection caused by the bacterium Streptococcus agalactiae (S. agalactiae) (also known as group B streptococcus or GBS). Group B streptococcal infection can cause serious illness and sometimes death, especially in newborns, the elderly, and people with compromised immune systems. GBS was recognized as a pathogen in cattle by Edmond Nocard and Mollereau in the late 1880s, but its significance as a human pathogen was not discovered before 1938, when Fry described three fatal cases of puerperal infections caused by GBS. In the early 1960s, GBS was recognized as a main cause of infections in newborns. In general, GBS is a harmless commensal bacterium being part of the human microbiota colonizing the gastrointestinal and genitourinary tracts of up to 30% of healthy human adults (asymptomatic carriers).S. agalactiae is also a common veterinary pathogen, because it can cause bovine mastitis (inflammation of the udder) in dairy cows. The species name "agalactiae" meaning "no milk", alludes to this.S. agalactiae is a Gram-positive coccus (spherical bacterium) with a tendency to form chains (streptococcus), beta-haemolytic, catalase-negative, and facultative anaerobe.S. agalactiae is the species designation for streptococci belonging to the group B of the Rebecca Lancefield classification of streptococci (Lancefield grouping). GBS is surrounded by a bacterial capsule composed of polysaccharides (exopolysaccharides). GBS are subclassified into 10 serotypes (Ia, Ib, II–IX) depending on the immunologic reactivity of their polysaccharide capsule. As other virulent bacteria, GBS harbours an important number of virulence factors, the most important are the capsular polysaccharide (rich in sialic acid), and a pore-forming toxin, β-haemolysin. The GBS capsule is probably the key virulence factor because it helps GBS escape from the host defence mechanisms interfering with phagocytic killing of GBS by human phagocytes. The GBS β-haemolysin is considered identical to the GBS pigment.

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